Exposure to high heat stimulates the production of heat shock proteins (HSPs), which play a critical role in repairing misfolded proteins, stabilizing mitochondrial membranes, and preventing oxidative damage under metabolic stress. These proteins help refold damaged cellular structures and activate autophagic pathways, ensuring dysfunctional mitochondria are recycled and replaced with more efficient counterparts. At the same time, sauna-induced hyperthermia triggers mitochondrial uncoupling prot